We all know that pesticides are needed to kill insects in case of crop pests. But how do pesticides kill insects? Why do some drugs have an immediate effect? Why do some drugs take two or three days to work? Why do you get insects in less than a week after taking the medicine? Why are some insects getting harder and harder to kill? To know why, you first need to understand the insecticidal mechanism and mode of action of pesticides. Only by understanding these two issues,SPRITEX insect killer supplier can we use pesticides scientifically, thus improving the effectiveness of pesticide control and economic benefits, and reducing the pollution of pesticides on the environment. The following editorial on this knowledge, through the collection and organization and everyone together to learn and discuss.
Insecticide mechanism
Insecticides against pests common site of action or physiological information systems designed to include through the neural network system and muscle, respiratory management system, growth environment regulation, midgut, unknown or non-specific five aspects.
1. Nervous system and muscle: acetylcholinesterase inhibitors,RAMBO insect killer manufacturer γ-aminobutyric acid chloride channel antagonists, sodium channel modulators, nicotinic acetylcholine receptor competitive modulators, nicotinic acetylcholine receptor modulators, glutamate-gated chloride channel modulators, voltage-dependent sodium channel blockers, and nicotinic receptor modulators for fish. The results showed that the insecticide could cause hyperexcitability, convulsions, lethargy, paralysis, paralysis, and muscle atrophy in the insect body. I believe this can simply be interpreted as death by excitement. These insecticides are usually fast-acting insecticides and quick-acting insecticides.
2. Respiratory system:Mitochondrial respiration produces adenosine triphosphate, which provides energy for all important cellular developmental processes. In mitochondria, the electron transport chain uses the energy released from oxidation to form a proton electrochemical gradient that drives the synthesis of adenosine triphosphate.Chinese VITFE Insecticide spray This is a bit of a mouthful, and can be simply understood as a lack of cellular energy and starvation of insects. Insecticides that act on a single target in the system usually have a moderate or fast kill rate.
3. Growth regulation: Insect development is controlled by a balance of two major hormones, the juvenile hormone and the ecdysteroid hormone. Insect growth regulators work by mimicking one of these hormones or by directly affecting epidermal deposition or lipid biosynthesis. Juvenile hormones interfere with or prevent insect metamorphosis, which can be interpreted as developmental arrest, growth inhibition, and asphyxiation. Ecdysteroids induce premature molting and accelerate the molting process, which can be interpreted as premature molting and shedding of the skin to death. In this system, insecticides acting on a single target are usually slow acting.
4. midgut:Lepidoptera-specific microbial toxins sprayed or expressed in transgenic crops. Includes insect midgut microbes that disrupt protein toxins that bind to receptors on the midgut membrane to form openings, causing ionic imbalance and sepsis; baculovirus granulomatous viruses, and nucleopolyhedroviruses. Can be understood as death by poisoning. Insecticides that act on a single target in the system usually work at a moderate rate.
5. Unknown and non-specific: compounds whose mechanism of action and impact is unknown or undetermined by research, unknown bacterial biological agents, unknown fungal agents. Can be directly understood as an inexplicable death. The speed of action development varies from species to species.
Mode of action of insecticides
In addition to their own toxicity, insecticides must first invade the insect in some way, then reach the site of action, and finally act at the target site to kill or repel the insect. This way of invading the insect body, that is, the mode of action of insecticides, including contact, stomach poison, inhalation, fumigation and so on.
1. Touch kill: drug and insect epidermis or tarsus contact, can penetrate the body wall into the body to reach the site of action, causing insect poisoning death. This mode of action is called contact killing. A kind of drug with touch killing effect is called touch killing agent. Examples are phoxim, parathion, deltamethrin and methomyl. The main factors affecting poisoning by touch are the structure of the insect's epidermis and the physical and chemical properties of the touch agent.
2. Gastrotoxicity: The way the drug enters the gastrointestinal system with the insect's oral organs and is absorbed by the midgut, and reaches the functional parts through the circulatory system to cause insect poisoning and kill the function is called gastrotoxic function. Drugs known as gastric toxins have gastric toxicity, such as organophosphorus chemical, pyrethroid pesticides. The gastric toxicity of gastric toxicity should have the following two conditions: insect feeding does not cause vomiting or diarrhea, the drug with the food into the digestive tract can be dissolved and absorbed.
3. Endosorption: Pesticides are absorbed by the roots, stems and leaves of plants, and can be transported or transformed into more toxic substances in the plant. Poisoning caused by insects eating toxic stems and leaves is called systemic effects. It mainly refers to the endosorbents used for the prevention and treatment of stinging mouthparts, and sometimes this mode of action is categorized as gastric toxicity.
4. Fumigation: the gas of toxic substances produced by the vaporization of insecticides can enter our body by studying the respiratory management system of insects and make the
